- An anaplerotic approach to correct the mitochondrial dysfunction in ataxia-telangiectasia (A-T)
- Increased susceptibility of airway epithelial cells from ataxia-telangiectasia to S. pneumoniae infection due to oxidative damage and impaired innate immunity.
- Loss of ATM in Airway Epithelial Cells Is Associated with Susceptibility to Oxidative Stress.
- Metabolic Stress and Mitochondrial Dysfunction in Ataxia-Telangiectasia
- Senataxin controls meiotic silencing through ATR activation and chromatin remodeling.
